Pathological report of four patients presenting with cranial dystonias
Identifieur interne : 006532 ( Main/Exploration ); précédent : 006531; suivant : 006533Pathological report of four patients presenting with cranial dystonias
Auteurs : Gibb [Royaume-Uni] ; Andrew Lees (neurologue) [Royaume-Uni] ; C. D. Marsden [Royaume-Uni]Source :
- Movement Disorders [ 0885-3185 ] ; 1988.
English descriptors
- KwdEn :
- Aged, Angioma, Basal ganglia, Blepharospasm, Blepharospasm (pathology), Brain (pathology), Brain Neoplasms (complications), Brain Stem (pathology), Central tegmental tract, Dystonia, Dystonia (etiology), Dystonia (pathology), Dystonia (physiopathology), Female, Hemangioma (complications), Humans, Male, Mandible (physiopathology), Middle Aged, Mouth (physiopathology), Pons.
- MESH :
- complications : Brain Neoplasms, Hemangioma.
- etiology : Dystonia.
- pathology : Blepharospasm, Brain, Brain Stem, Dystonia.
- physiopathology : Dystonia, Mandible, Mouth.
- Aged, Female, Humans, Male, Middle Aged.
Abstract
Secondary dystonias and experimental models of dystonia suggest that mechanisms responsible for primary dystonias may lie in the basal gaglia or brainstem. A histological study ahs been done in three patients with cranial dystonia (blepharospasm with oromandibular dystonia in two, blepharospasm alone in one), and one patient with craniocervical dystonia (oromandibular dystonia with retrocollis). In the patient with blepharospasm alone, an angioma, 0.5 mm in diameter, was found in the dorsal pons at the site of the central tegmental tract, confirming that some patients presenting with primary dystonias may have longstanding lesions in the brainstem. In the three other cases, the striatum, pallidum, thalamus, and brainstem were examined and cell populations in the putamen, substantia nigra, and inferior olives were compared with age‐matched controls, but no significant abnormality was found.
Url:
DOI: 10.1002/mds.870030305
Affiliations:
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J." last="Lees">Andrew Lees (neurologue)</name><affiliation wicri:level="3"><country>Royaume-Uni</country><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName><wicri:orgArea>Department of Neuropathology, National Hospitals for Nervous Diseases, Maida Vale</wicri:orgArea><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName><orgName>National Hospital for Neurology and Neurosurgery</orgName></affiliation></author><author><name sortKey="Marsden, C D" sort="Marsden, C D" uniqKey="Marsden C" first="C. D." last="Marsden">C. D. Marsden</name><affiliation wicri:level="2"><country>Royaume-Uni</country><placeName><region type="country">Angleterre</region></placeName><wicri:cityArea>University Department of Clinical Neurology, The National Hospital, London</wicri:cityArea></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="sub">Official Journal of the Movement Disorder Society</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1988">1988</date><biblScope unit="vol">3</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="211">211</biblScope><biblScope unit="page" to="221">221</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">EB90B87ED41BC408D204584464684C77AD719D69</idno><idno type="DOI">10.1002/mds.870030305</idno><idno type="ArticleID">MDS870030305</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Angioma</term><term>Basal ganglia</term><term>Blepharospasm</term><term>Blepharospasm (pathology)</term><term>Brain (pathology)</term><term>Brain Neoplasms (complications)</term><term>Brain Stem (pathology)</term><term>Central tegmental tract</term><term>Dystonia</term><term>Dystonia (etiology)</term><term>Dystonia (pathology)</term><term>Dystonia (physiopathology)</term><term>Female</term><term>Hemangioma (complications)</term><term>Humans</term><term>Male</term><term>Mandible (physiopathology)</term><term>Middle Aged</term><term>Mouth (physiopathology)</term><term>Pons</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Brain Neoplasms</term><term>Hemangioma</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Blepharospasm</term><term>Brain</term><term>Brain Stem</term><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Dystonia</term><term>Mandible</term><term>Mouth</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Secondary dystonias and experimental models of dystonia suggest that mechanisms responsible for primary dystonias may lie in the basal gaglia or brainstem. A histological study ahs been done in three patients with cranial dystonia (blepharospasm with oromandibular dystonia in two, blepharospasm alone in one), and one patient with craniocervical dystonia (oromandibular dystonia with retrocollis). In the patient with blepharospasm alone, an angioma, 0.5 mm in diameter, was found in the dorsal pons at the site of the central tegmental tract, confirming that some patients presenting with primary dystonias may have longstanding lesions in the brainstem. In the three other cases, the striatum, pallidum, thalamus, and brainstem were examined and cell populations in the putamen, substantia nigra, and inferior olives were compared with age‐matched controls, but no significant abnormality was found.</div></front></TEI><affiliations><list><country><li>Royaume-Uni</li></country><region><li>Angleterre</li><li>Grand Londres</li></region><settlement><li>Londres</li></settlement><orgName><li>National Hospital for Neurology and Neurosurgery</li></orgName></list><tree><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Gibb" sort="Gibb" uniqKey="Gibb" last="Gibb">Gibb</name></region><name sortKey="Lees, A J" sort="Lees, A J" uniqKey="Lees A" first="A. J." last="Lees">Andrew Lees (neurologue)</name><name sortKey="Marsden, C D" sort="Marsden, C D" uniqKey="Marsden C" first="C. D." last="Marsden">C. D. Marsden</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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